для тех, у кого генетический анализ показывает мутацию гена, регулирующего ферменты, которые неактивную форму витамина д3 превращают в активную форму, этот препарат номер 1. Долгое время витамин D… Читать ещё
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Serova","avatarId":"20706/enc-3302fffa092310a6570d9cd16c1bc69f","url":"/user/4hrzg42ejxf67j2kw91k9bu2gr?utm_source=Hum&main_tab=Hum","level":" ","verified":false,"linkTheme":"normal","scoreQuestion":"","publicId":"4hrzg42ejxf67j2kw91k9bu2gr","reviewsCount":62},"rating":{"newDesign":false,"max":5,"val":5},"reactions":{"likesCount":0,"dislikesCount":0,"trustedLikesCount":0,"myTrustedLike":false,"myReaction":"none"},"photos":[],"moderated":true,"blocked":false,"time":"28 января","text":"для тех, у кого генетический анализ показывает мутацию гена, регулирующего ферменты, которые неактивную форму витамина д3 превращают в активную форму, этот препарат номер 1. Долгое время витамин D считался «витамином для костей». В ряде экспериментальных моделей показано, что активный витамин D ослабляет сигнальные пути, которые часто перегружены в опухолях. Один из них — путь Wnt/β-катенин, важный для бесконтрольного роста клеток. Когда этот путь становится менее активным, деление замедляется, а клетки чаще возвращаются под нормальный контроль.\n\nЭто не делает витамин D лекарством от рака. Но объясняет, почему дефицит витамина D часто связан с более агрессивным течением болезни и худшим прогнозом. Его роль здесь не в «убийстве» опухоли, а в тонкой настройке клеточных сигналов, от которых зависит баланс между ростом и сдерживанием. Feldman D., Krishnan A.V. et al., Nature Reviews Cancer, 2014.\nDOI: 10.1038/nrc3691","fullText":"","reviewContra":"","reviewPro":"","reviewComment":"для тех, у кого генетический анализ показывает мутацию гена, регулирующего ферменты, которые неактивную форму витамина д3 превращают в активную форму, этот препарат номер 1. Долгое время витамин D считался «витамином для костей». В ряде экспериментальных моделей показано, что активный витамин D ослабляет сигнальные пути, которые часто перегружены в опухолях. Один из них — путь Wnt/β-катенин, важный для бесконтрольного роста клеток. Когда этот путь становится менее активным, деление замедляется, а клетки чаще возвращаются под нормальный контроль.\n\nЭто не делает витамин D лекарством от рака. Но объясняет, почему дефицит витамина D часто связан с более агрессивным течением болезни и худшим прогнозом. Его роль здесь не в «убийстве» опухоли, а в тонкой настройке клеточных сигналов, от которых зависит баланс между ростом и сдерживанием. Feldman D., Krishnan A.V. et al., Nature Reviews Cancer, 2014.\nDOI: 10.1038/nrc3691","textTranslations":{"EN":"for those who have a genetic analysis showing a mutation in the gene that regulates enzymes that convert the inactive form of vitamin d3 into the active form, this drug is number 1. For a long time, vitamin D was considered a \"vitamin for bones.\" A number of experimental models have shown that active vitamin D weakens signaling pathways that are often overloaded in tumors. One of them is the Wnt/β—catenin pathway, which is important for uncontrolled cell growth. When this pathway becomes less active, division slows down, and cells return to normal control more often.\n\nThis does not make vitamin D a cure for cancer. But it explains why vitamin D deficiency is often associated with a more aggressive course of the disease and a worse prognosis. Its role here is not to \"kill\" the tumor, but to fine-tune the cellular signals on which the balance between growth and containment depends. Feldman D., Krishnan A.V. et al., Nature Reviews Cancer, 2014.\nDOI: 10.1038/nrc3691","TR":"olanlar için genetik analiz gösterir gen mutasyonu, düzenleyici enzimler devre dışı formu vitamin d3 açmak aktif bir formu, bu ilacın 1 numara. Uzun bir süre D vitamini «olarak kabul edildi vitamini kemikler için». Bazı deneysel modellerde gösterilmiştir aktif D vitamini zayıflatır sinyal yolları, sık sık bunalmış tümörler. Bunlardan biri yolu Wnt/β-катенин için önemli kontrolsüz hücre büyümesi. Bu yol daha az aktif hale geldiğinde bölünme yavaşlar ve hücreler daha sık normal kontrole döner.\n\nBu, D vitaminini kanser ilacı yapmaz. Ancak D vitamini eksikliğinin neden genellikle hastalığın daha agresif seyrine ve daha kötü prognoza bağlı olduğunu açıklıyor. Onun rolü, burada değil, «cinayet» tümör ve ince ayar hücresel sinyallerin bağlı olduğu arasında bir denge büyüme ve muhafaza. 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